Primary Site >> Liver Cancer

Gene >> MGAT3

  • 1998
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  • 2017
Ref: Progression of hepatic neoplasms is severely retarded in mice lacking the bisecting N-acetylglucosamine on N-glycans: evidence for a glycoprotein factor that facilitates hepatic tumor progression.
PMID: 9661906
Ref: New evidence for an extra-hepatic role of N-acetylglucosaminyltransferase III in the progression of diethylnitrosamine-induced liver tumors in mice.
PMID: 10866326
Ref: Truncated, inactive N-acetylglucosaminyltransferase III (GlcNAc-TIII) induces neurological and other traits absent in mice that lack GlcNAc-TIII.
PMID: 11986323
Ref: The bisecting GlcNAc on N-glycans inhibits growth factor signaling and retards mammary tumor progression.
PMID: 20395209
Ref: Cell surface glycan alterations in epithelial mesenchymal transition process of Huh7 hepatocellular carcinoma cell.
PMID: 23977005
Ref: Expression of N-Acetylglucosaminyltransferase III Suppresses alpha2,3-Sialylation, and Its Distinctive Functions in Cell Migration Are Attributed to alpha2,6-Sialylation Levels.
PMID: 26801611
Ref: DNA hypomethylation upregulates expression of the MGAT3 gene in HepG2 cells and leads to changes in N-glycosylation of secreted glycoproteins.
PMID: 27073020
Ref: Reduced N-acetylglucosaminyltransferase III expression via Smad3 and Erk signaling in TGF-beta1-induced HCC EMT model.
PMID: 28245423