ViMRT
}  
 
Home   
< Docs   

 HBV mutation literature information.


  Mutations in the HBV PreS/S gene related to hepatocellular carcinoma in Vietnamese chronic HBV-infected patients.
 PMID: 35390033       2022       PloS one
Table: S167L


  Effect of hepatitis B virus (HBV) surface-gene variability on markers of replication during treated human immunodeficiency virus-HBV infection in Western Africa.
 PMID: 30257068       2019       Liver international
Abstract: Twelve S-gene MUPIQHs were identified among 21 patients (28.8%): sS140L (n = 4), sD144A (n = 1), sS167L (n = 2), sS174N (n = 6), sP178Q (n = 2), sG185L (n = 2), sW191L (n = 2), sP203Q/R (n = 2), sS204N/I/R/K/T/G (n = 7), sN207T


  Nearly half of Ultrio plus NAT non-discriminated reactive blood donors were identified as occult HBV infection in South China.
 PMID: 31269905       2019       BMC infectious diseases
Discussion: Some mutations such as Q101R, S167 L, V168A, R169H, S174 N, L175S, V177A, Q129R, D144E/A and G145A/R were in concordance with previous studies.


  Characterization of Novel Hepatitis B Virus PreS/S-Gene Mutations in a Patient with Occult Hepatitis B Virus Infection.
 PMID: 27182775       2016       PloS one
Table: S167L


  Serologic and genotypic characterization of hepatitis B virus in HIV-1 infected patients from South West and Littoral Regions of Cameroon.
 PMID: 27769271       2016       Virology journal
Table: S167L


  Occult hepatitis B virus infection in anti-HBs-positive infants born to HBsAg-positive mothers in China.
 PMID: 23951004       2013       PloS one
Discussion: identified the OBI-associated mutations Y100S, T116N, R122P, S143L and S167L from a large-sample, blood-donor study.


  Novel HBsAg markers tightly correlate with occult HBV infection and strongly affect HBsAg detection.
 PMID: 22086128       2012       Antiviral research
Abstract: By co-variation analysis, correlations were observed for R122P+S167L (phi=0.68, P=0.01), T116N+S143L (phi=0.53, P=0.03), and Y100S+S143L (phi=0.67, p<0.001).
Abstract: Mutants (obtained by site-directed mutagenesis) carrying T116N, T116N+S143L, R122P, R122P+Q101R, or R122P+S167L strongly decreased HBsAg-reactivity (54.9+-22.6S/CO, 31.2+-12.0S/CO, 6.1+-2.4S/CO, 3.0+-1.0S/CO and 3.9+-1.3S/CO, respectively) compared to wild-type (306.8+-64.1S/CO).


  Ultra-deep pyrosequencing detects conserved genomic sites and quantifies linkage of drug-resistant amino acid changes in the hepatitis B virus genome.
 PMID: 22666402       2012       PloS one
Result: These 20 variants did not include the immune escape variant sG145R, detected in relatively high frequencies at pre-treatment, but otherwise included the variants sS167L, sW172C, and sW172*, located at the minimal recognized sequence (positions s165 to s172) of the TH-s156/s175 epitope; among these, sS167L showed a continuous percentage increase from pre-treatment to ETV-VBK (sample 4A: 0.2%, 4B: 0.7%, 4C: 1.1%, 4D: 1.3%, 4E: 4%).
Discussion: In addition, the s



Browser Board

 Co-occurred Entities




   Filtrator